The 5'-terminal Non-coding Region of Murray Valley Encephalitis Virus RNA is Highly Conserved
نویسندگان
چکیده
منابع مشابه
The 5'-terminal non-coding region of Murray Valley encephalitis virus RNA is highly conserved.
The 5' non-coding region of the genomes of 11 isolates of Murray Valley encephalitis virus from Australia and Papua New Guinea were examined by primer extension sequencing. Although the 5' non-coding region of all isolates was found to be highly conserved, three isolates were significantly different in that they contained extra uridine residues. Two of these isolates from Papua New Guinea conta...
متن کاملMurray Valley encephalitis virus.
ANSW Public Health Officer Training Program, NSW Department of Health BArbovirus and Emerging Diseases Unit, Institute of Clinical Pathology and Medical Research, Westmead Hospital CDepartment of Medical Entomology, Institute of Clinical Pathology and Medical Research, Westmead Hospital mosquito samples and/or detecting seroconversion to MVEV in sentinel chicken flocks. These surveillance syste...
متن کاملPurification of Murray Valley encephalitis virus.
Methods are described for the purification of Murray Valley encephalitis virus from baby mouse brain extract. The method finally chosen involved treatment with protamine, ultracentrifugation, adsorption to and elution from hydroxyl apatite and a second ultracentrifugation. The degrees of recovery of virus infectivity and haemagglutinin were about 15 yo and 50 yo, respectively. Measurement of th...
متن کاملMR findings in Murray Valley encephalitis.
Murray Valley encephalitis (MVE) is caused by a flavivirus related to West Nile and St. Louis encephalitis viruses. We report a case of MVE resulting in quadriplegia and respiratory failure. MR imaging demonstrated thalamic hyperintensity on T2-weighted images, with similar involvement of the red nucleus, substantia nigra, and cervical cord. These findings preceded serologic diagnosis and are s...
متن کاملMechanism of virulence attenuation of glycosaminoglycan-binding variants of Japanese encephalitis virus and Murray Valley encephalitis virus.
The in vivo mechanism for virulence attenuation of laboratory-derived variants of two flaviviruses in the Japanese encephalitis virus (JEV) serocomplex is described. Host cell adaptation of JEV and Murray Valley encephalitis virus (MVE) by serial passage in adenocarcinoma cells selected for variants characterized by (i) a small plaque phenotype, (ii) increased affinity to heparin-Sepharose, (ii...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Journal of General Virology
سال: 1990
ISSN: 0022-1317,1465-2099
DOI: 10.1099/0022-1317-71-1-241